Solving the pathogenesis of ankylosing spondylitis

(2018) Solving the pathogenesis of ankylosing spondylitis. Clinical Immunology, 186, pp. 46-50.

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Description

• HLA-B associations of AS are complex, with protective and risk associations in addition to the associations with HLA-B27 having been established. • Interaction between ERAP1 and HLA-B27 and -B40 has been demonstrated, indicating that ERAP1 works probably by similar mechanisms, with these genes to cause AS. • AS appears to be driven by interaction between the gut microbiome and host immune system offering the potential to treat the condition by gut targeted therapies. • HLA-B27 forms homodimers, and there is strong evidence that these operate in AS to induce AS through interactions with KIR bearing immune cells.

Impact and interest:

6 citations in Scopus
7 citations in Web of Science®
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ID Code: 223109
Item Type: Contribution to Journal (Journal Article)
Refereed: Yes
ORCID iD:
Brown, Mattorcid.org/0000-0003-0538-8211
Measurements or Duration: 5 pages
Keywords: Aminopeptidase genes and AS, ERAP1, HLA-B associations, HLA-B27, HLA-B40, ankylosing spondylitis
DOI: 10.1016/j.clim.2017.07.011
ISSN: 1521-6616
Pure ID: 33324173
Divisions: Past > QUT Faculties & Divisions > Faculty of Health
Past > Institutes > Institute of Health and Biomedical Innovation
Copyright Owner: Consult author(s) regarding copyright matters
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Deposited On: 07 Nov 2021 03:37
Last Modified: 01 Nov 2025 18:47